The ability of mutations to cause cancer depends on how fast they force cells to divide, Sinai Health researchers have found.

When these switches were applied to colon cancer cells, the cells reverted to a normal-like state. These results were then confirmed by molecular and cellular experiments performed on animal subjects.

The risk of cancer goes up with age, in part because aging impedes the body's ability to detect and destroy cells with ...

Johns Hopkins Medicine scientists say they have found a pattern of so-called epigenetic "marks" in a transition state between normal and pancreatic cancer cells in mice, and that the normal cells may ...

One of the challenges of fighting pancreatic cancer is finding ways to penetrate the organ's dense tissue to define the margins between malignant and normal tissue. A new study uses DNA origami ...